Objects
Waters, David W., Schuliga, Michael, Mutsaers, Steven E., Grainge, Christopher L., Knight, Darryl A., Pathinayake, Prabuddha S., Wei, Lan, Tan, Hui-Ying, Blokland, Kaj E. C., Jaffar, Jade, Westall, Glen P., Burgess, Janette K., Prele, Cecilia M.. MDPI AG; 2021. A senescence bystander effect in human lung fibroblasts.
Blokland, Kaj E. C., Waters, David W., Burgess, Janette K., Knight, Darryl A., Schuliga, Michael, Read, Jane, Pouwels, Simon D., Grainge, Christopher L., Jaffar, Jade, Westall, Glen, Mutsaers, Steven E., Prele, Cecilia M.. MDPI AG; 2020. Senescence of IPF lung fibroblasts disrupt alveolar epithelial cell proliferation and promote migration in wound healing.
Waters, David W., Blokland, Kaj E. C., Pathinayake, Prabuddha S., Wei, Lan, Schuliga, Michael, Jaffar, Jade, Westall, Glen P., Hansbro, Philip M., Prele, Cecilia M., Mutsaers, Steven E., Bartlett, Nathan W., Burgess, Janette K., Grainge, Christopher L., Knight, Darryl A.. American Thoracic Society; 2019. STAT3 regulates the onset of oxidant-induced senescence in lung fibroblasts.
Schuliga, Michael, Jaffar, Jade, Stewart, Alastair G., Berhan, Asres, Langenbach, Shenna, Harris, Trudi, Waters, David, Lee, Peter V. S., Grainge, Christopher, Westall, Glen, Knight, Darryl. American Physiological Society; 2017. Annexin A2 contributes to lung injury and fibrosis by augmenting factor Xa fibrogenic activity.
Schuliga, Michael, Read, Jane, Blokland, Kaj E. C., Waters, David W., Burgess, Janette, Prele, Cecilia, Mutsaers, Steven E., Jaffar, Jade, Westall, Glen, Reid, Andrew, James, Allen, Grainge, Christopher, Knight, Darryl A.. Portland Press; 2020. Self DNA perpetuates IPF lung fibroblast senescence in a cGAS-dependent manner.
Schuliga, Michael, Pechkovsky, Dmitri V., Mutsaers, Steven E., Jaffar, Jade, Westall, Glen, Grainge, Christopher, Knight, Darryl A., Read, Jane, Waters, David W., Blokland, Kaj E. C., Reid, Andrew T., Hogaboam, Cory M., Khalil, Nasreen, Burgess, Janette K., Prêle, Cecilia M.. Wiley-Blackwell; 2018. Mitochondrial dysfunction contributes to the senescent phenotype of IPF lung fibroblasts.